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Everyday Noises Can Hurt Hearts, Not Just Ears, and the Ability to Learn

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Tuesday, April 16, 2024

Ten years ago Jamie Banks started working from her home in the town of ­Lincoln, Mass. After a couple of months, the continuing racket from landscaping machines began to feel unendurable, even when she was inside her home. “This horrible noise was going on for hours every day, every week—leaf blowers, industrial lawnmowers, hedge trimmers,” she says. The sound of a gas-powered leaf blower outside can be as loud as 75 decibels (dB) to someone listening from inside a house—higher than the World Health Organization cutoff to protect hearing over a 24-hour period. “I started thinking, this can’t be good,” she says. “It’s definitely not good for me. It certainly can’t be good for the workers operating the equipment. And there are lots of kids and lots of seniors around. It can’t be good for them either.”Banks is a health-care specialist and environmental scientist who has worked most of her life as a consultant on health outcomes and behavior change for government agencies, law firms and corporations. She decided to do something about her situation and got together with a like-minded neighbor to pester the town government. It took the pair seven years to get their town to do one thing—ban gas-powered leaf blowers during the summer. The process was long and frustrating, and it made Banks think about going bigger and helping others.So she did. In June 2023 Quiet Communities, a nonprofit group that Banks founded and runs, sued the U.S. Environmental Protection Agency for not publishing or enforcing rules and regulations to limit loud sounds: unmuffled motorcycles, cacophonous factories, the thunder of an airplane just overhead, the roar of an elevated train, the scream of a sound­track in a spin class, headphones set too loud. There is a federal law that calls for the EPA to do this, but it hasn’t been enforced for more than 40 years.On supporting science journalismIf you're enjoying this article, consider supporting our award-winning journalism by subscribing. By purchasing a subscription you are helping to ensure the future of impactful stories about the discoveries and ideas shaping our world today.Banks’s idea that loud noise “can’t be good” is well supported by science. Noise can damage more than just your ears. Through daytime stress and nighttime sleep disturbances, loud sounds can hurt your heart and blood vessels, disrupt your endocrine system, and make it difficult to think and learn. The World Health Organization calculated that in 2018 in the European Union, 1.6 million years of healthy life were lost because of traffic noise. The organization recommended that to avoid these health effects, exposure to road traffic noise should be limited to below a weighted 24-hour average of 53 dB (the sound of a campfire from about 16 feet away) during the day, evening, and night and 45 dB specifically at night (the sound of light traffic about 100 feet away).Precise “safe” levels to avoid specific ailments are hard to come by. But in general, research shows, reducing loud noise can reduce the risk of harm. There are several ways to protect yourself. Various organizations have made maps that indicate quiet and noisy places around the U.S. Smartphone apps can tell you if you’re in one that’s too loud for safety. And noise experts all seem to own earbuds and headphones and use them often to block out the din.For most of human history, the issue with noise was simply how annoying it can be. The first noise ordinance on record was drafted by Julius Caesar shortly before his assassination in 44 B.C.E., limiting the times that noisy carts and wagons could be on the street. The modern industrial era brought regulations to protect the ears of workers exposed to steam engines, drop forges, and other loud machinery but little information or action on everyday noises. A big moment came in 1970, when psychoacoustics expert Karl Kryter, then at the Stanford Research Institute, published The Effects of Noise on Man. The book focused on what loud sound could do to hearing and touched on work performance, sleep, vision and blood circulation.That noise has biological effects beyond the ear makes sense in evolutionary terms. Noise may signal that a herd of elephants is charging your compound or that a pack of wolves is close by—you need to know, and your body needs to get ready for something unpleasant. As noise and sleep researcher Mathias Basner of the University of Pennsylvania and his colleagues put it in a 2014 Lancet review, “evolution has programmed human beings to be aware of sounds as possible sources of danger.”MSJONESNYC; Source: Centers for Disease Control and Prevention (reference)From an evolutionary point of view, sleep was “a very dangerous stage,” a time when you had to maintain attention to your environment, Basner says. But the psychiatrist and epidemiologist, who has spent much of his career studying the effects of airport noise on people sleeping nearby, notes a “watchman function” that leads to night awakenings is for the most part harmful, not helpful, in modern societies.A lot of people think they sleep soundly despite nearby noise. They should think again. Basner has exposed hundreds of people to noise during sleep studies. He says many would get up in the morning swearing they’d slept through the night without waking, but the data showed they’d had num­er­ous awakenings.By the early 1970s a poll showed that the public considered noise pollution a serious problem. Formal government recognition came in 1972 with the passage of the Noise Control Act and the establishment of the EPA’s Office of Noise Abatement and Control. The act promised that the government would “promote an environment for all Americans free from noise that jeopardizes their health or welfare.” At the time, the EPA estimated that 100 million Americans experienced daily average sound of 55 dB or over. Fifty-­five dB is about halfway between the level of a quiet conversation at home and one in a restaurant or office. Any 24-hour exposure average louder than that, according to the EPA, was loud enough to interfere with activities and cause annoyance.By this time, studies from universities in the U.S. and Europe were beginning to identify health effects of noise beyond the ear, starting with behavior and learning. In 1973 three U.S. researchers, with funding from the National Science Foundation and two private organizations, studied 73 children in primary school who lived in several 32-story apartment buildings clustered over Interstate 95 where it passes through New York City. Children on the lower floors, exposed to more highway noise, were less able to distinguish sounds and were reading at a lower level than children on the higher floors. There was even a dose-response relation: the longer the child had lived in the building, the lower their scores were likely to be.In 1975 researchers at the City University of New York looked at school records for 161 primary school students at a school that was 220 feet from an elevated subway, with trains hurtling by every 4.5 minutes. The records showed a three- to four-month reading lag for kids in classrooms on the noisy side of the building compared with those in classes on the quiet side.Researchers were able to do a natural ex­­per­i­ment when the Munich International Airport moved about 25 miles north in 1992. The scientists found that among children living near the old airport site, long-term memory and reading skills improved after the airport closed. But for kids near the new airport, those changes went in the opposite direction, and their stress hormone levels increased.In the early 2000s Stephen Stansfeld, then a psychiatrist at the University of London, studied kids aged nine to 11 living and going to school near airports in Europe, comparing their blood pressure and learning ability with those of similar children who did not live under flight paths. Airplane noise reached 77 dB(A) at several schools; dB(A) is a decibel scale that em­­phasizes frequencies the human ear hears best. “We found a straight-line relationship between increasing levels of aircraft noise and children’s reading comprehension,” Stansfeld says. “Noisy schools were not healthy educational environments.” A colleague found the harmful effects lasted into secondary school.All the while, the U.S. was getting noisier. In 2014 Rick Neitzel, an environmental and occupational health professor at the University of Michigan who has been researching noise for 25 years, and his colleagues estimated that more than 100 million Americans had a continuous average exposure level in 24 hours of greater than 70 dB. Imagine standing next to a washing machine all day or suffering occasional blasts from the gas-powered lawn equipment Jamie Banks could hear inside her house. It was a rise of 15 dB in just a generation, which is the difference between normal conversation and a vacuum cleaner.Beyond the brain and cognition, the heart and blood vessels also take a hit from noise—perhaps not surprising given the stressful effects of noise and the impacts of stress on the circulatory system. A slew of epidemiological studies over the years have linked environmental noise, especially nighttime noise, to high blood pressure, heart failure, myocardial infarction (heart attacks) and stroke. The association held true even after researchers controlled for confounders such as air pollution and socioeconomic variables.Some of the strongest human data come from Denmark, which is an epidemiologist’s dream country because it collects health data on pretty much every resident. Mette Sørensen, an epidemiologist at Ros­kilde University in Denmark, Thomas Münzel, a professor at Johannes Gutenberg University in Germany, and others teased apart the effects of noise on types of heart disease such as myocardial in­­farc­tion, angina and heart failure. Looking at 2.5 million people 50 years or older, they found road traffic noise increased the incidence of all three. In a 2021 report on 3.6 million Danes, they showed that an average daily 10-dB increase in sound exposure because of road noise increased the risk of stroke by 3 to 4 percent.They’ve also looked at type 2 diabetes, a condition that had already been associated with chronic sleep disturbance. This link makes sense, Sørensen says: stress such as frequent awakening raises levels of glucocorticoids, which inhibit insulin secretion and insulin sensitivity. Reducing these two things leads to diabetes. In 2013 Sørensen and her colleagues re­­port­ed an 8 percent increase in diabetes risk for every 10-dB increase in exposure to road traffic noise. Eight years later, looking at 3.56 million Danes 35 years and older, with 233,912 new cases of diabetes, they calculated that road traffic noise could be blamed for 8.5 percent of the cases of diabetes in Denmark and railway noises for 1.4 percent.Sørensen is aware that those percentages don’t sound very high. But they are meaningful, she says. In Denmark, more than one third of the population is exposed to average daily sound levels above 58 dB. “You have such a huge proportion exposed to this,” she says, “so even though it’s only a really small in­­crease in risk, it’s a large number of people who get diabetes due to noise.”The physical mechanisms behind these links are still being investigated, but animal studies have highlighted possible culprits. (Researchers cannot deliberately expose people to such potentially harmful noise effects.) Münzel explored some of these connections in mice, for example. In one study, he ex­­posed the rodents to average sound levels of 72 dB over four days and found that the animals had higher blood pressure and levels of stress hormones and inflammation, as well as changes in the activity of genes that regulate vascular health and cell death.Jen Christiansen; Source: “Environmental Noise and the Cardiovascular System,” by Thomas Münzel et al., in Journal of the American College of Cardiology, Vol. 71; February 2018 (reference)In the U.S., most research on noise has been done without much help from the federal government, despite the Noise Control Act. In 1981, after Ronald Reagan was elected president on a promise of cutting back the federal government, he appointed Anne Gorsuch as head of the EPA; she eliminated funding for the agency’s noise-control office. “She wanted to show the White House that she believed in small government,” says Sidney Shapiro, a Wake Forest administrative law professor who has studied the rise and fall of noise-abatement laws. He says noise has never had a well-organized constituency to support it. Responsibility for noise-control research, funding and regulation was left to individual state and local governments.Today the EPA’s noise-control office is still there—on paper. “There is no money to enforce regulations or for research or education,” Neitzel says. That’s why Quiet Communities is suing. “Not having the EPA doing its job is hugely damaging, not only to the public who are being harmed by noise but also to the research community. We don’t have access to a stream of funding that should be there.”Without that information, noise researchers have long struggled to quantify the overall impact of the American din. In 2014 when Neitzel and his colleagues at the University of Michigan wanted to figure out whether reducing noise would have a beneficial effect on cardiovascular disease, they had to resort to prevalence estimates made in 1981. In 2015 they published their findings. A 5-dB reduction in average noise exposure would cut the prevalence of high blood pressure by 1.5 percent and cut heart disease by 1.8 percent. Again, these are low numbers. But because of the high incidence of these conditions to begin with, an average 5-dB reduction would have an annual economic benefit of $3.9 billion. “I was shocked that the numbers were as big as they were,” Neitzel says.Overall, as with chemical and air pollution, people with lower incomes are being hit the hardest. Their communities may have highways running through them or have factories and airports nearby. “Folks who are already in marginalized communities may be bearing way more than their fair share of noise exposure,” Neitzel says.In these areas, it’s essential to ground research and solutions in community priorities, says Erica Walker, an epidemiologist at Brown University. Walker founded the Community Noise Lab, which works with communities to study and mitigate the effects of noise and other pollutants. She believes that it’s probably not just the absolute sound level that determines bodily damage—it’s unwanted sound. If the sound is a welcome one, does prolonged exposure to, say, 75 dB (about the volume of street musicians playing trumpets 30 feet away from you) raise stress levels the way that large studies have shown? “We need to know what the difference is between sound and noise from an individual point of view and from a community perspective,” Walker says.She points to the Shaw neighborhood of Washington, D.C., which has been undergoing gentrification. “The cultural practice was to play go-go music. As the neighborhood began to become gentrified, newcomers had their own acoustical expectations of what the neighborhood should sound like,” Walker says. “If I’m going into a community and I’m measuring noise and I’m saying it’s really loud (based strictly on decibels) and harmful to health, that might be a misclassification.” People already in the community might perceive that noise as comfortable.Walker and her colleagues are now trying to tease apart unacceptable noise and acceptable sound. In an ongoing study, they’ve been asking volunteers how they feel about different kinds of noise. Then the researchers deconstruct those noises by rearranging them, making them unidentifiable as a specific sound but maintaining the decibel level and frequency spectrum (think high notes and low notes). By the end of this summer, Walker hopes to know whether the deconstructed sound matches up with the recognizable sound. Such information could help distinguish the roles of sound intensity and cultural connotation in hu­­man harm.Whatever your community’s sound tolerance, you can protect yourself from noise that’s intolerable. The simplest way, of course, is to avoid it. Sørensen’s data show that sleeping on the quieter side of a building, away from the street, makes a difference. Or you can move to a quieter area. That is easier said than done, and all the experts I spoke with noted that moving to a more peaceful place, as many of them have, is possible only for people who can afford it. If you plan to move, Basner advises visiting the new area at different times of day.For noise that can’t be avoided, science may offer some promise, at least for ear effects. Sudden loud noises (think concerts, jet engines, leaf blowers and loud machines) stimulate the delicate hair cells and nerve fibers in the inner ear, resulting in the release of damaging free radicals. Animal work has identified some promising chemicals to sop these molecules up, says Colleen Le Prell, a psychologist and head of the department of speech, language and hearing at the University of Texas at Dallas, who is working on several candidates. There is already a drug for children to prevent chemotherapy-induced hearing loss, but it has significant side effects and isn’t approved for general use.The Montello Foundation’s artist retreat in Nevada has been identified by the nonprofit Quiet Parks International as a community without irritating noise.If you want to get a snapshot of the sound around you, the Internet can help. The National Institute for Occupational Health and Safety has a national map, but it works only on Apple mobile devices right now. The U.S. Department of Transportation has a map for transportation noise, but it doesn’t include workplace noise or inside noise. You can see noise across the entire country, albeit at pretty low resolution, on a National Park Service sound map.To measure sound directly, there are plenty of smartphone apps. Don’t be surprised if the numbers are high. Data from Apple watches suggest that one in three adult Americans is exposed to excessive noise and daily averages of 70 dB(A) (the sound of an older washing machine or dishwasher) or greater. Those levels are considered by both the World Health Organization and the EPA as dangerous to the ear. You can see state-by-state results on Apple Hearing Study U.S. maps. Apple watches and iPhones can be set to alert you when sound reaches a particular level.The data collected from Apple watches come from the Apple Hearing Study, begun in 2019 by Neitzel and his colleagues at the University of Michigan and funded by Apple. The study shows that a quieter world is possible. It took the lockdowns of COVID to prove it. The researchers got smartphone data from about 6,000 volunteers, covering a period from just before the pandemic began in January 2020 through late April of that year, when many businesses and activities had shut down for safety, and lots of people were staying close to home. The data showed a 3-dB(A) drop in noise exposure. Because decibels are measured on a logarithmic scale, that’s a halving of sound energy, easily noticeable by the human ear.Sørensen moved from a city out into the country and checked a noise map first. Neitzel is very intentional about his exposure. “One thing that I absolutely try to do is make sure I’ve programmed periods into the day that I’m not going to have noise exposure,” he says. That means a bike ride through a quiet area or turning the TV off. If he’s at a bus stop, he stands back from the street as much as he can, and he routinely wears noise-blocking earplugs or earmuffs—sometimes both—when he’s checking out industrial sites.Neitzel protects his ears at concerts as well. “There’s a bit of social stigma around wearing ear protection at a concert,” he says, so he wears clear plugs, much like many musicians use. And he’s got noise-canceling headphones and earbuds. They seal the ear to limit outside sound, which permits listening at a lower volume. He and his family wear noise-canceling earbuds on planes.You can ask others to turn sound down. Sharon Kujawa, an audiologist at Massachusetts Eye and Ear hospital in Boston, and her colleagues did an experiment to see whether people in spin classes preferred louder or softer sound. They liked softer. The facility managers were reluctant to make a change, but eventually customer requests got them to agree to a 3-dB decrease in volume. Fellow ear researcher Le Prell had her children use volume-limiting headphones. The kids were in marching bands in high school, in the percussion section, and she donated earplugs to the entire group.As for specific levels to aim for, that’s a tough one. There’s no formula that says x hours of exposure to road traffic noise will raise your risk of heart disease y percent. The EPA, which established its noise standards in 1974, before the full health effects were so clear, indicates that a 24-hour exposure level of 70 dB or less will prevent any hearing damage, and 55 dB outdoors and 45 dB indoors will prevent activity interference and annoyance. For lack of anything more current, that’s the standard used by many noise researchers today.In terms of protective devices, there are only limited federal regulations on headphones, and there’s some concern that the devices go up to volumes that can damage the health (ear and otherwise) of children. Volume limiters on headphones generally have an upper limit of 85 dB, but what the limit should really be, and for how long, is anybody’s guess. There’s also no solid research on whether devices that produce masking noises help.Clear, consistent standards for how much is too much, and what works, are unlikely without a revitalization of the EPA’s noise-control office. An agency spokesperson wouldn’t say whether the lawsuit by Quiet Communities will spur any change. The two sides in the suit “are currently in the midst of filing motions and cross-motions,” says Quiet Communities lawyer Sanne Knudsen of the University of Washington. When we spoke, Knudsen expected some kind of agreement would be reached by April and hoped it would be one that got the Office of Noise Abatement and Control up and running again.Jamie Banks now spends most of her time in a quiet town in rural Maine, which, she says, is blissfully free of loud lawn equipment and other noise. She is optimistic that a newly active federal noise-control office will establish data-based noise limits and regulations and that the EPA will ensure regulations are enforced. In 1972, when the noise office was established, the Los Angeles Times opined that it wouldn’t mean an instant reduction in harmful sound, “but at least a start has been made.” Fifty-two years later Banks hopes for not just a start but real progress.

Experts describe ways to turn down the volume, from earbuds to smartphone apps that detect harmful noise levels

Ten years ago Jamie Banks started working from her home in the town of ­Lincoln, Mass. After a couple of months, the continuing racket from landscaping machines began to feel unendurable, even when she was inside her home. “This horrible noise was going on for hours every day, every week—leaf blowers, industrial lawnmowers, hedge trimmers,” she says. The sound of a gas-powered leaf blower outside can be as loud as 75 decibels (dB) to someone listening from inside a house—higher than the World Health Organization cutoff to protect hearing over a 24-hour period. “I started thinking, this can’t be good,” she says. “It’s definitely not good for me. It certainly can’t be good for the workers operating the equipment. And there are lots of kids and lots of seniors around. It can’t be good for them either.”

Banks is a health-care specialist and environmental scientist who has worked most of her life as a consultant on health outcomes and behavior change for government agencies, law firms and corporations. She decided to do something about her situation and got together with a like-minded neighbor to pester the town government. It took the pair seven years to get their town to do one thing—ban gas-powered leaf blowers during the summer. The process was long and frustrating, and it made Banks think about going bigger and helping others.

So she did. In June 2023 Quiet Communities, a nonprofit group that Banks founded and runs, sued the U.S. Environmental Protection Agency for not publishing or enforcing rules and regulations to limit loud sounds: unmuffled motorcycles, cacophonous factories, the thunder of an airplane just overhead, the roar of an elevated train, the scream of a sound­track in a spin class, headphones set too loud. There is a federal law that calls for the EPA to do this, but it hasn’t been enforced for more than 40 years.


On supporting science journalism

If you're enjoying this article, consider supporting our award-winning journalism by subscribing. By purchasing a subscription you are helping to ensure the future of impactful stories about the discoveries and ideas shaping our world today.


Banks’s idea that loud noise “can’t be good” is well supported by science. Noise can damage more than just your ears. Through daytime stress and nighttime sleep disturbances, loud sounds can hurt your heart and blood vessels, disrupt your endocrine system, and make it difficult to think and learn. The World Health Organization calculated that in 2018 in the European Union, 1.6 million years of healthy life were lost because of traffic noise. The organization recommended that to avoid these health effects, exposure to road traffic noise should be limited to below a weighted 24-hour average of 53 dB (the sound of a campfire from about 16 feet away) during the day, evening, and night and 45 dB specifically at night (the sound of light traffic about 100 feet away).

Precise “safe” levels to avoid specific ailments are hard to come by. But in general, research shows, reducing loud noise can reduce the risk of harm. There are several ways to protect yourself. Various organizations have made maps that indicate quiet and noisy places around the U.S. Smartphone apps can tell you if you’re in one that’s too loud for safety. And noise experts all seem to own earbuds and headphones and use them often to block out the din.

For most of human history, the issue with noise was simply how annoying it can be. The first noise ordinance on record was drafted by Julius Caesar shortly before his assassination in 44 B.C.E., limiting the times that noisy carts and wagons could be on the street. The modern industrial era brought regulations to protect the ears of workers exposed to steam engines, drop forges, and other loud machinery but little information or action on everyday noises. A big moment came in 1970, when psychoacoustics expert Karl Kryter, then at the Stanford Research Institute, published The Effects of Noise on Man. The book focused on what loud sound could do to hearing and touched on work performance, sleep, vision and blood circulation.

That noise has biological effects beyond the ear makes sense in evolutionary terms. Noise may signal that a herd of elephants is charging your compound or that a pack of wolves is close by—you need to know, and your body needs to get ready for something unpleasant. As noise and sleep researcher Mathias Basner of the University of Pennsylvania and his colleagues put it in a 2014 Lancet review, “evolution has programmed human beings to be aware of sounds as possible sources of danger.”

Bar chart shows sounds measured in decibels. Household appliances reach about 70 dB. 2 hours of listening to 95 dB—the noise of a motorcycle—may damage hearing. That can happen after 5 minutes of noise between 105 and 110 dB, the sound of a loud radio.

MSJONESNYC; Source: Centers for Disease Control and Prevention (reference)

From an evolutionary point of view, sleep was “a very dangerous stage,” a time when you had to maintain attention to your environment, Basner says. But the psychiatrist and epidemiologist, who has spent much of his career studying the effects of airport noise on people sleeping nearby, notes a “watchman function” that leads to night awakenings is for the most part harmful, not helpful, in modern societies.

A lot of people think they sleep soundly despite nearby noise. They should think again. Basner has exposed hundreds of people to noise during sleep studies. He says many would get up in the morning swearing they’d slept through the night without waking, but the data showed they’d had num­er­ous awakenings.

By the early 1970s a poll showed that the public considered noise pollution a serious problem. Formal government recognition came in 1972 with the passage of the Noise Control Act and the establishment of the EPA’s Office of Noise Abatement and Control. The act promised that the government would “promote an environment for all Americans free from noise that jeopardizes their health or welfare.” At the time, the EPA estimated that 100 million Americans experienced daily average sound of 55 dB or over. Fifty-­five dB is about halfway between the level of a quiet conversation at home and one in a restaurant or office. Any 24-hour exposure average louder than that, according to the EPA, was loud enough to interfere with activities and cause annoyance.

By this time, studies from universities in the U.S. and Europe were beginning to identify health effects of noise beyond the ear, starting with behavior and learning. In 1973 three U.S. researchers, with funding from the National Science Foundation and two private organizations, studied 73 children in primary school who lived in several 32-story apartment buildings clustered over Interstate 95 where it passes through New York City. Children on the lower floors, exposed to more highway noise, were less able to distinguish sounds and were reading at a lower level than children on the higher floors. There was even a dose-response relation: the longer the child had lived in the building, the lower their scores were likely to be.

In 1975 researchers at the City University of New York looked at school records for 161 primary school students at a school that was 220 feet from an elevated subway, with trains hurtling by every 4.5 minutes. The records showed a three- to four-month reading lag for kids in classrooms on the noisy side of the building compared with those in classes on the quiet side.

Researchers were able to do a natural ex­­per­i­ment when the Munich International Airport moved about 25 miles north in 1992. The scientists found that among children living near the old airport site, long-term memory and reading skills improved after the airport closed. But for kids near the new airport, those changes went in the opposite direction, and their stress hormone levels increased.

In the early 2000s Stephen Stansfeld, then a psychiatrist at the University of London, studied kids aged nine to 11 living and going to school near airports in Europe, comparing their blood pressure and learning ability with those of similar children who did not live under flight paths. Airplane noise reached 77 dB(A) at several schools; dB(A) is a decibel scale that em­­phasizes frequencies the human ear hears best. “We found a straight-line relationship between increasing levels of aircraft noise and children’s reading comprehension,” Stansfeld says. “Noisy schools were not healthy educational environments.” A colleague found the harmful effects lasted into secondary school.

All the while, the U.S. was getting noisier. In 2014 Rick Neitzel, an environmental and occupational health professor at the University of Michigan who has been researching noise for 25 years, and his colleagues estimated that more than 100 million Americans had a continuous average exposure level in 24 hours of greater than 70 dB. Imagine standing next to a washing machine all day or suffering occasional blasts from the gas-powered lawn equipment Jamie Banks could hear inside her house. It was a rise of 15 dB in just a generation, which is the difference between normal conversation and a vacuum cleaner.

Beyond the brain and cognition, the heart and blood vessels also take a hit from noise—perhaps not surprising given the stressful effects of noise and the impacts of stress on the circulatory system. A slew of epidemiological studies over the years have linked environmental noise, especially nighttime noise, to high blood pressure, heart failure, myocardial infarction (heart attacks) and stroke. The association held true even after researchers controlled for confounders such as air pollution and socioeconomic variables.

Some of the strongest human data come from Denmark, which is an epidemiologist’s dream country because it collects health data on pretty much every resident. Mette Sørensen, an epidemiologist at Ros­kilde University in Denmark, Thomas Münzel, a professor at Johannes Gutenberg University in Germany, and others teased apart the effects of noise on types of heart disease such as myocardial in­­farc­tion, angina and heart failure. Looking at 2.5 million people 50 years or older, they found road traffic noise increased the incidence of all three. In a 2021 report on 3.6 million Danes, they showed that an average daily 10-dB increase in sound exposure because of road noise increased the risk of stroke by 3 to 4 percent.

They’ve also looked at type 2 diabetes, a condition that had already been associated with chronic sleep disturbance. This link makes sense, Sørensen says: stress such as frequent awakening raises levels of glucocorticoids, which inhibit insulin secretion and insulin sensitivity. Reducing these two things leads to diabetes. In 2013 Sørensen and her colleagues re­­port­ed an 8 percent increase in diabetes risk for every 10-dB increase in exposure to road traffic noise. Eight years later, looking at 3.56 million Danes 35 years and older, with 233,912 new cases of diabetes, they calculated that road traffic noise could be blamed for 8.5 percent of the cases of diabetes in Denmark and railway noises for 1.4 percent.

Sørensen is aware that those percentages don’t sound very high. But they are meaningful, she says. In Denmark, more than one third of the population is exposed to average daily sound levels above 58 dB. “You have such a huge proportion exposed to this,” she says, “so even though it’s only a really small in­­crease in risk, it’s a large number of people who get diabetes due to noise.”

The physical mechanisms behind these links are still being investigated, but animal studies have highlighted possible culprits. (Researchers cannot deliberately expose people to such potentially harmful noise effects.) Münzel explored some of these connections in mice, for example. In one study, he ex­­posed the rodents to average sound levels of 72 dB over four days and found that the animals had higher blood pressure and levels of stress hormones and inflammation, as well as changes in the activity of genes that regulate vascular health and cell death.

Flow chart shows a model of how environmental noise may be linked to increased risk of disease–from exposure, to stress and inflammation, to diseased states.

Jen Christiansen; Source: “Environmental Noise and the Cardiovascular System,” by Thomas Münzel et al., in Journal of the American College of Cardiology, Vol. 71; February 2018 (reference)

In the U.S., most research on noise has been done without much help from the federal government, despite the Noise Control Act. In 1981, after Ronald Reagan was elected president on a promise of cutting back the federal government, he appointed Anne Gorsuch as head of the EPA; she eliminated funding for the agency’s noise-control office. “She wanted to show the White House that she believed in small government,” says Sidney Shapiro, a Wake Forest administrative law professor who has studied the rise and fall of noise-abatement laws. He says noise has never had a well-organized constituency to support it. Responsibility for noise-control research, funding and regulation was left to individual state and local governments.

Today the EPA’s noise-control office is still there—on paper. “There is no money to enforce regulations or for research or education,” Neitzel says. That’s why Quiet Communities is suing. “Not having the EPA doing its job is hugely damaging, not only to the public who are being harmed by noise but also to the research community. We don’t have access to a stream of funding that should be there.”

Without that information, noise researchers have long struggled to quantify the overall impact of the American din. In 2014 when Neitzel and his colleagues at the University of Michigan wanted to figure out whether reducing noise would have a beneficial effect on cardiovascular disease, they had to resort to prevalence estimates made in 1981. In 2015 they published their findings. A 5-dB reduction in average noise exposure would cut the prevalence of high blood pressure by 1.5 percent and cut heart disease by 1.8 percent. Again, these are low numbers. But because of the high incidence of these conditions to begin with, an average 5-dB reduction would have an annual economic benefit of $3.9 billion. “I was shocked that the numbers were as big as they were,” Neitzel says.

Overall, as with chemical and air pollution, people with lower incomes are being hit the hardest. Their communities may have highways running through them or have factories and airports nearby. “Folks who are already in marginalized communities may be bearing way more than their fair share of noise exposure,” Neitzel says.

In these areas, it’s essential to ground research and solutions in community priorities, says Erica Walker, an epidemiologist at Brown University. Walker founded the Community Noise Lab, which works with communities to study and mitigate the effects of noise and other pollutants. She believes that it’s probably not just the absolute sound level that determines bodily damage—it’s unwanted sound. If the sound is a welcome one, does prolonged exposure to, say, 75 dB (about the volume of street musicians playing trumpets 30 feet away from you) raise stress levels the way that large studies have shown? “We need to know what the difference is between sound and noise from an individual point of view and from a community perspective,” Walker says.

She points to the Shaw neighborhood of Washington, D.C., which has been undergoing gentrification. “The cultural practice was to play go-go music. As the neighborhood began to become gentrified, newcomers had their own acoustical expectations of what the neighborhood should sound like,” Walker says. “If I’m going into a community and I’m measuring noise and I’m saying it’s really loud (based strictly on decibels) and harmful to health, that might be a misclassification.” People already in the community might perceive that noise as comfortable.

Walker and her colleagues are now trying to tease apart unacceptable noise and acceptable sound. In an ongoing study, they’ve been asking volunteers how they feel about different kinds of noise. Then the researchers deconstruct those noises by rearranging them, making them unidentifiable as a specific sound but maintaining the decibel level and frequency spectrum (think high notes and low notes). By the end of this summer, Walker hopes to know whether the deconstructed sound matches up with the recognizable sound. Such information could help distinguish the roles of sound intensity and cultural connotation in hu­­man harm.

Whatever your community’s sound tolerance, you can protect yourself from noise that’s intolerable. The simplest way, of course, is to avoid it. Sørensen’s data show that sleeping on the quieter side of a building, away from the street, makes a difference. Or you can move to a quieter area. That is easier said than done, and all the experts I spoke with noted that moving to a more peaceful place, as many of them have, is possible only for people who can afford it. If you plan to move, Basner advises visiting the new area at different times of day.

For noise that can’t be avoided, science may offer some promise, at least for ear effects. Sudden loud noises (think concerts, jet engines, leaf blowers and loud machines) stimulate the delicate hair cells and nerve fibers in the inner ear, resulting in the release of damaging free radicals. Animal work has identified some promising chemicals to sop these molecules up, says Colleen Le Prell, a psychologist and head of the department of speech, language and hearing at the University of Texas at Dallas, who is working on several candidates. There is already a drug for children to prevent chemotherapy-induced hearing loss, but it has significant side effects and isn’t approved for general use.

Desert landscape with building on the right and mountain in the background.

The Montello Foundation’s artist retreat in Nevada has been identified by the nonprofit Quiet Parks International as a community without irritating noise.

If you want to get a snapshot of the sound around you, the Internet can help. The National Institute for Occupational Health and Safety has a national map, but it works only on Apple mobile devices right now. The U.S. Department of Transportation has a map for transportation noise, but it doesn’t include workplace noise or inside noise. You can see noise across the entire country, albeit at pretty low resolution, on a National Park Service sound map.

To measure sound directly, there are plenty of smartphone apps. Don’t be surprised if the numbers are high. Data from Apple watches suggest that one in three adult Americans is exposed to excessive noise and daily averages of 70 dB(A) (the sound of an older washing machine or dishwasher) or greater. Those levels are considered by both the World Health Organization and the EPA as dangerous to the ear. You can see state-by-state results on Apple Hearing Study U.S. maps. Apple watches and iPhones can be set to alert you when sound reaches a particular level.

The data collected from Apple watches come from the Apple Hearing Study, begun in 2019 by Neitzel and his colleagues at the University of Michigan and funded by Apple. The study shows that a quieter world is possible. It took the lockdowns of COVID to prove it. The researchers got smartphone data from about 6,000 volunteers, covering a period from just before the pandemic began in January 2020 through late April of that year, when many businesses and activities had shut down for safety, and lots of people were staying close to home. The data showed a 3-dB(A) drop in noise exposure. Because decibels are measured on a logarithmic scale, that’s a halving of sound energy, easily noticeable by the human ear.

Sørensen moved from a city out into the country and checked a noise map first. Neitzel is very intentional about his exposure. “One thing that I absolutely try to do is make sure I’ve programmed periods into the day that I’m not going to have noise exposure,” he says. That means a bike ride through a quiet area or turning the TV off. If he’s at a bus stop, he stands back from the street as much as he can, and he routinely wears noise-blocking earplugs or earmuffs—sometimes both—when he’s checking out industrial sites.

Neitzel protects his ears at concerts as well. “There’s a bit of social stigma around wearing ear protection at a concert,” he says, so he wears clear plugs, much like many musicians use. And he’s got noise-canceling headphones and earbuds. They seal the ear to limit outside sound, which permits listening at a lower volume. He and his family wear noise-canceling earbuds on planes.

You can ask others to turn sound down. Sharon Kujawa, an audiologist at Massachusetts Eye and Ear hospital in Boston, and her colleagues did an experiment to see whether people in spin classes preferred louder or softer sound. They liked softer. The facility managers were reluctant to make a change, but eventually customer requests got them to agree to a 3-dB decrease in volume. Fellow ear researcher Le Prell had her children use volume-limiting headphones. The kids were in marching bands in high school, in the percussion section, and she donated earplugs to the entire group.

As for specific levels to aim for, that’s a tough one. There’s no formula that says x hours of exposure to road traffic noise will raise your risk of heart disease y percent. The EPA, which established its noise standards in 1974, before the full health effects were so clear, indicates that a 24-hour exposure level of 70 dB or less will prevent any hearing damage, and 55 dB outdoors and 45 dB indoors will prevent activity interference and annoyance. For lack of anything more current, that’s the standard used by many noise researchers today.

In terms of protective devices, there are only limited federal regulations on headphones, and there’s some concern that the devices go up to volumes that can damage the health (ear and otherwise) of children. Volume limiters on headphones generally have an upper limit of 85 dB, but what the limit should really be, and for how long, is anybody’s guess. There’s also no solid research on whether devices that produce masking noises help.

Clear, consistent standards for how much is too much, and what works, are unlikely without a revitalization of the EPA’s noise-control office. An agency spokesperson wouldn’t say whether the lawsuit by Quiet Communities will spur any change. The two sides in the suit “are currently in the midst of filing motions and cross-motions,” says Quiet Communities lawyer Sanne Knudsen of the University of Washington. When we spoke, Knudsen expected some kind of agreement would be reached by April and hoped it would be one that got the Office of Noise Abatement and Control up and running again.

Jamie Banks now spends most of her time in a quiet town in rural Maine, which, she says, is blissfully free of loud lawn equipment and other noise. She is optimistic that a newly active federal noise-control office will establish data-based noise limits and regulations and that the EPA will ensure regulations are enforced. In 1972, when the noise office was established, the Los Angeles Times opined that it wouldn’t mean an instant reduction in harmful sound, “but at least a start has been made.” Fifty-two years later Banks hopes for not just a start but real progress.

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In Alaska’s Warming Arctic, Photos Show an Indigenous Elder Passing Down Hunting Traditions

An Inupiaq elder teaches his great-grandson to hunt in rapidly warming Northwest Alaska where thinning ice, shifting caribou migrations and severe storms are reshaping life

KOTZEBUE, Alaska (AP) — The low autumn light turned the tundra gold as James Schaeffer, 7, and his cousin Charles Gallahorn, 10, raced down a dirt path by the cemetery on the edge of town. Permafrost thaw had buckled the ground, tilting wooden cross grave markers sideways. The boys took turns smashing slabs of ice that had formed in puddles across the warped road.Their great-grandfather, Roswell Schaeffer, 78, trailed behind. What was a playground to the kids was, for Schaeffer – an Inupiaq elder and prolific hunter – a reminder of what warming temperatures had undone: the stable ice he once hunted seals on, the permafrost cellars that kept food frozen all summer, the salmon runs and caribou migrations that once defined the seasons.Now another pressure loomed. A 211-mile mining road that would cut through caribou and salmon habitat was approved by the Trump administration this fall, though the project still faces lawsuits and opposition from environmental and native groups. Schaeffer and other critics worry it could open the region to outside hunters and further devastate already declining herds. “If we lose our caribou – both from climate change and overhunting – we’ll never be the same,” he said. “We’re going to lose our culture totally.”Still, Schaeffer insists on taking the next generation out on the land, even when the animals don’t come. It was late September and he and James would normally have been at their camp hunting caribou. But the herd has been migrating later each year and still hadn’t arrived – a pattern scientists link to climate change, mostly caused by the burning of oil, gas and coal. So instead of caribou, they scanned the tundra for swans, ptarmigan and ducks.Caribou antlers are stacked outside Schaeffer's home. Traditional seal hooks and whale harpoons hang in his hunting shed. Inside, a photograph of him with a hunted beluga is mounted on the wall beside the head of a dall sheep and a traditional mask his daughter Aakatchaq made from caribou hide and lynx fur.He got his first caribou at 14 and began taking his own children out at 7. James made his first caribou kill this past spring with a .22 rifle. He teaches James what his father taught him: that power comes from giving food and a hunter’s responsibility is to feed the elders.“When you’re raised an Inupiaq, your whole being is to make sure the elders have food,” he said.But even as he passes down those lessons, Schaeffer worries there won’t be enough to sustain the next generation – or to sustain him. “The reason I’ve been a successful hunter is the firm belief that, when I become old, people will feed me,” he said. “My great-grandson and my grandson are my future for food.” That future feels tenuous These days, they’re eating less hunted food and relying more on farmed chicken and processed goods from the store. The caribou are fewer, the salmon scarcer, the storms more severe. Record rainfall battered Northwest Alaska this year, flooding Schaeffer’s backyard twice this fall alone. He worries about the toll on wildlife and whether his grandchildren will be able to live in Kotzebue as the changes accelerate.“It’s kind of scary to think about what’s going to happen,” he said.That afternoon, James ducked into the bed of Schaeffer’s truck and aimed into the water. He shot two ducks. Schaeffer helped him into waders – waterproof overalls – so they could collect them and bring them home for dinner, but the tide was too high. They had to turn back without collecting the ducks. The changes weigh on others, too. Schaeffer’s friend, writer and commercial fisherman Seth Kantner grew up along the Kobuk River, where caribou once reliably crossed by the hundreds of thousands. “I can hardly stand how lonely it feels without all the caribou that used to be here,” he said. “This road is the largest threat. But right beside it is climate change.”The Associated Press receives support from the Walton Family Foundation for coverage of water and environmental policy. The AP is solely responsible for all content. For all of AP’s environmental coverage, visit https://apnews.com/hub/climate-and-environmentCopyright 2025 The Associated Press. All rights reserved. This material may not be published, broadcast, rewritten or redistributed.Photos You Should See – December 2025

Changes to polar bear DNA could help them adapt to global heating, study finds

Scientists say bears in southern Greenland differ genetically to those in the north, suggesting they could adjustChanges in polar bear DNA that could help the animals adapt to warmer climates have been detected by researchers, in a study thought to be the first time a statistically significant link has been found between rising temperatures and changing DNA in a wild mammal species.Climate breakdown is threatening the survival of polar bears. Two-thirds of them are expected to have disappeared by 2050 as their icy habitat melts and the weather becomes hotter. Continue reading...

Changes in polar bear DNA that could help the animals adapt to warmer climates have been detected by researchers, in a study thought to be the first time a statistically significant link has been found between rising temperatures and changing DNA in a wild mammal species.Climate breakdown is threatening the survival of polar bears. Two-thirds of them are expected to have disappeared by 2050 as their icy habitat melts and the weather becomes hotter.Now scientists at the University of East Anglia have found that some genes related to heat stress, ageing and metabolism are behaving differently in polar bears living in south-east Greenland, suggesting they may be adjusting to warmer conditions.The researchers analysed blood samples taken from polar bears in two regions of Greenland and compared “jumping genes”: small, mobile pieces of the genome that can influence how other genes work. Scientists looked at the genes in relation to temperatures in the two regions and at the associated changes in gene expression.“DNA is the instruction book inside every cell, guiding how an organism grows and develops,” said the lead researcher, Dr Alice Godden. “By comparing these bears’ active genes to local climate data, we found that rising temperatures appear to be driving a dramatic increase in the activity of jumping genes within the south-east Greenland bears’ DNA.”As local climates and diets evolve as a result of changes in habitat and prey forced by global heating, the genetics of the bears appear to be adapting, with the group of bears in the warmest part of the country showing more changes than the communities farther north. The authors of the study have said these changes could help us understand how polar bears might survive in a warming world, inform understanding of which populations are most at risk and guide future conservation efforts.This is because the findings, published on Friday in the journal Mobile DNA, suggest the genes that are changing play a crucial role in how different polar bear populations are evolving.Godden said: “This finding is important because it shows, for the first time, that a unique group of polar bears in the warmest part of Greenland are using ‘jumping genes’ to rapidly rewrite their own DNA, which might be a desperate survival mechanism against melting sea ice.”Temperatures in north-east Greenland are colder and less variable, while in the south-east there is a much warmer and less icy environment, with steep temperature fluctuations.DNA sequences in animals change over time, but this process can be accelerated by environmental stress such as a rapidly heating climate.There were some interesting DNA changes, such as in areas linked to fat processing, that could help polar bears survive when food is scarce. Bears in warmer regions had more rough, plant-based diets compared with the fatty, seal-based diets of northern bears, and the DNA of south-eastern bears seemed to be adapting to this.Godden said: “We identified several genetic hotspots where these jumping genes were highly active, with some located in the protein-coding regions of the genome, suggesting that the bears are undergoing rapid, fundamental genetic changes as they adapt to their disappearing sea ice habitat.”The next step will be to look at other polar bear populations, of which there are 20 around the world, to see if similar changes are happening to their DNA.This research could help protect the bears from extinction. But the scientists said it was crucial to stop temperature rises accelerating by reducing the burning of fossil fuels.Godden said: “We cannot be complacent, this offers some hope but does not mean that polar bears are at any less risk of extinction. We still need to be doing everything we can to reduce global carbon emissions and slow temperature increases.”

A Deadly Pathogen Decimated Sunflower Sea Stars. Look Inside the Lab Working to Bring Them Back by Freezing and Thawing Their Larvae

For the first time, scientists have cryopreserved and revived the larvae of a sea star species. The breakthrough, made with the giant pink star, gives hope the technique could be repeated to save the imperiled predator

A Deadly Pathogen Decimated Sunflower Sea Stars. Look Inside the Lab Working to Bring Them Back by Freezing and Thawing Their Larvae For the first time, scientists have cryopreserved and revived the larvae of a sea star species. The breakthrough, made with the giant pink star, gives hope the technique could be repeated to save the imperiled predator Juvenile sunflower sea stars at the Sunflower Star Laboratory in Moss Landing, California. At this phase, each is less than an inch wide, but they can grow to be more than three feet across as adults. Avery Schuyler Nunn Key takeaways: Recovering sunflower sea stars by freezing them in time Ravaged by infectious bacteria, sunflower sea stars literally wasted away across the Pacific coast of North America—and their resulting population crash destabilized kelp forest ecosystems. Scientists pioneered a cryopreservation technique on the closely related giant pink star, raising hopes that a bank of frozen sunflower star larvae could one day be thawed in the same way and released into the wild. Along a working California harbor, where gulls wheel over weathered pilings and the old Western Flyer—the ship John Steinbeck once sailed to the Sea of Cortez—sits restored in its berth, researchers buzz about in a modest lab tucked between warehouses and boatyards. Inside, amid the hiss of pumps and the faint smell of brine from seawater tables, a scientist lifts a small vial from a plume of liquid nitrogen, its frosted casing holding the tiniest flicker of hope for a species on the brink. Each of the 18 vials contains between 500 and 700 larval giant pink sea stars. At this stage, they are tiny specks suspended in seawater, invisible to the naked eye. These particular larvae have been cryopreserved and stored at roughly minus 180 degrees Celsius since March. At the Sunflower Star Laboratory (SSL) in Moss Landing, California, scientists thawed the larval pink sea stars and coaxed them to successfully develop into juveniles this summer—a first for any sea star species. In October, the scientists thawed another batch of larvae from the same cohort to test larval growth and survival under different freezing conditions and thawing protocols. The breakthrough, however, isn’t really about the giant pink star, a species that’s common in the wild. Instead, these larvae serve as a crucial stand-in for the far more imperiled sunflower sea star (Pycnopodia helianthoides)—a vanishing species for which larvae are precious, limited and increasingly difficult to obtain. Perfecting cryopreservation methods on pink stars—ensuring they can survive freezing, resume feeding and grow into juveniles—lays the scientific groundwork for facilitating a return of Pycnopodia. The contents of a thawed vial are placed under a microscope to assess viability of the larvae. Avery Schuyler Nunn The discovery arrives at a precarious time, as sunflower stars have disappeared at a pace rarely seen in marine ecosystems. As a mysterious pathogen ravaged their population along the western shores of North America beginning in 2013, the creatures collapsed from an estimated six billion individuals to functional extinction in parts of their range—all within just a few years. Their loss left kelp forests with dramatically fewer predators, destabilizing ecosystems across the Pacific coast and allowing urchins to proliferate and graze formerly lush underwater canopies into barren rock. Now, scientists hope that “freezing” their larvae will offer a new avenue for bringing the species back. “Cryopreservation is particularly important on the population level when thinking about recovery for this endangered species, because it had major population losses,” says Marissa Baskett, an environmental scientist at the University of California, Davis, who was not involved in the project. The process lets scientists preserve the sea stars’ existing genetic diversity for future reintroduction to the wild, she adds. “Especially given the uncertainty about different disease outbreaks, having that stock to return to is incredibly valuable.” A mysterious and “complete collapse” Sunflower sea stars have long lived in abundance up and down the rugged Pacific coast—from Alaskan archipelagoes to Baja California. The 24-limbed echinoderms sprawled across the seafloor in shades of ochre, crimson and violet. Among the fastest-moving and largest of all sea stars—capable of stretching nearly three feet across—these radiant predators coursed through kelp forests, voraciously hunting purple sea urchins and preventing them from over-grazing on the holdfasts that root towering golden canopies of kelp. An adult sunflower sea star has 24 limbs and can be more than three feet wide. This one was photographed off Point Dume State Beach near Los Angeles. Brent Durand via Getty Images “In Northern California and Oregon, there historically would have been multiple keystone predators within the kelp forest ecosystem who are punching on purple urchins and keeping their population in check,” says Reuven Bank, board chair of SSL. “But the southern sea otter was extirpated across its historic range, so we were left with sunflower stars being the last major keystone predator of purple urchins across over 100 miles of coastline.” “And sunflower stars didn’t just eat urchins, they scared them,” Bank adds. “Urchins can smell a sunflower star approaching, and in healthy kelp forests they hide more and graze less. Even without consuming them, sunflower stars helped keep urchin behavior, and therefore kelp forests, in balance.” Then, in June 2013, tidepool monitors along Washington’s Olympic Peninsula documented an unprecedented sight. The once-sturdy sea stars had turned soft, pale and contorted, their arms curling and detaching from their bodies. By late summer, the same mysterious affliction had surfaced in British Columbia, and it began sweeping both north and south with startling speed. The emerging epidemic, which caused the invertebrates to literally disintegrate, would soon be known as sea star wasting disease. An infamous marine heatwave—nicknamed “The Blob”—had settled over the Pacific by 2014, thrusting the coast into a fever. Ocean temperatures spiked, likely speeding up the disease progression in already stressed sea stars and leading to higher mortality. In the warm, stagnant water, infected sunflower stars dissolved at an eerily rapid pace, leaving behind ghost-white films of bacterial mass where the vibrant predators had been just days before. “You’d have apparently healthy stars basically melt away into puddles of goo within 48 hours,” says Andrew Kim, lab manager at SSL. “It happened so quickly, and I don’t think folks were prepared for the ensuing ecosystem shift. You don’t often expect diseases to come through and totally reshape ecosystem dynamics within such a short period. But that’s what we saw.” Without sunflower sea stars to keep those spiny purple urchins in check, the balance began to falter, setting the stage for an unprecedented chain reaction. Urchin populations skyrocketed, grazing on kelp without limits, and once-thriving underwater forests collapsed into barren rock. A dense group of purple sea urchins, which exploded in population after the sunflower sea stars disappeared, photographed near Mendocino Headlands State Park, north of San Francisco. Brent Durand via Getty Images In California, with 99 percent loss, sunflower sea stars are now considered functionally extinct. “Even though there may be a few remnant individuals left, they can no longer fulfill their historic role in the ecosystem,” Bank says. As sunflower stars unraveled in the wild, another species—its thick-armed cousin, the giant pink star—offered an unexpected foothold for hope. The pink stars share a nearly identical geographic range and life history with sunflower stars, and crucially, their larvae can be raised in aquaria. If scientists could learn to freeze and revive the pink star in its early life stages, they wondered, could that knowledge become a lifeline for the sunflower star? That’s where the small team in Moss Landing stepped in. Freezing sea stars for the future What these scientists did was something no one had ever pulled off with a sea star. Working with giant pink stars, researchers spawned adults at the Aquarium of the Pacific in Long Beach, California, fertilized their gametes to produce thousands of larvae, and shipped those microscopic bodies to the Frozen Zoo—a cryopreserved archive of creatures operated by the San Diego Zoo Wildlife Alliance. There, reproductive scientists plunged the larvae into liquid nitrogen, cooling them to extremely low temperatures and pausing their cells’ biological activity. The larvae, essentially frozen in time, were shielded from ice crystal damage with special cryoprotectant mixtures. Sunflower Star Laboratory researchers remove a vial of pink star larvae from an insulated cooler at around minus 180 degrees Celsius in preparation for thawing. Avery Schuyler Nunn After months in this suspended state, the larvae were sent to the Sunflower Star Laboratory where Carly Young, a San Diego Zoo Wildlife Alliance scientist who advances cryopreservation and reproductive-rescue tools, led the team in thawing the vials. She had fine-tuned the ideal way to keep the larvae alive as they returned to real-world temperatures, carefully testing more than 100 “recipes” with various warming rates, cryoprotectant dilutions and rehydration steps. The pink star larvae not only survived thawing, but have thus far lived all the way through metamorphosis into juveniles. Scientists watched the little stars settle spontaneously along the bottom of their beakers just 19 days after revival. The success prompted the team to apply the same cryopreservation protocols to sunflower star larvae from the Alaska SeaLife Center. The larvae will be frozen in perpetuity, creating the first-ever cryopreserved archive of the species—like a seed bank, but for the baby sea stars. “A famous quote from the ’70s, when the Frozen Zoo in San Diego was established, was, ‘You must collect things for reasons you don’t yet understand,’” says Ashley Kidd, conservation project manager at SSL. “We don’t know when the other shoe is going to drop and what populations are going to look like as the planet changes. So, rather than chasing ghosts around the ocean floor, we really focused on what we can do with animals that are currently under human care somewhere.” While cryopreservation itself isn’t a ready-made restoration tool, it opens the door to conserving genetic diversity of a species and banking rare lineages for potential reintroduction to the wild. In the 1970s and 1990s, researchers began testing cryopreservation of marine invertebrates with sperm and larvae, establishing the basic protocols that this team could apply to sea stars. The breakthrough doesn’t restore kelp forests by itself, but the SSL scientists note that cryopreservation creates something the conservation community has desperately needed: time. Time to hold onto genetic diversity, time to refine captive rearing and time to prepare for future reintroduction at scales big enough to matter. The ultimate test, the researchers say, will be translating the thawing process to sunflower sea stars. Carly Young, at the Sunflower Star Laboratory, looks for movement in the young sea stars. Avery Schuyler Nunn Just this summer, scientists uncovered a piece of the puzzle that had eluded them for more than a decade: the pathogen behind sea star wasting disease. In a four-year international effort, researchers traced the outbreak to a strain of the marine bacterium Vibrio pectenicida. When cultured and injected into healthy sea stars, it reproduced the telltale symptoms—softening arms, rapid disintegration and death within days. The finding, published in Nature Ecology and Evolution in August, gives recovery teams a way to test for the pathogen in labs and hatcheries, tighten quarantine measures and understand disease risks before returning captive-bred sea stars to the Pacific. “It’s massively important to know what to look for, and the fact that we are now able to test for this disease is going to be critical in advancing our ability to move forward with reintroductions and continuing the research,” notes Kim. “We’ve already been able to take fluid samples from all of our stars and get them analyzed for the presence of Vibrio pectenicida, so we’ve mobilized very quickly on the heels of development.” Paired with this new diagnostic clarity, advances in cryopreservation offer a second front in the effort to save the species. Frozen larvae can be stored for decades and offer flexibility for selective breeding of disease-tolerant traits, notes the team. Cryopreservation adds another tool to the scientists’ toolbox as they fight to prevent the species—and, in turn, its ecosystem—from wasting away. “Bringing back sunflower stars,” Bank says, “is the single-most important step we can take toward restoring kelp forest balance.” Get the latest Science stories in your inbox.

Archaeologists Are Unraveling the Mysteries Behind Deep Pits Found Near Stonehenge

Based on a comprehensive study, researchers are now convinced the shafts were human-made, likely dug during the Late Neolithic period roughly 4,000 years ago

Archaeologists Are Unraveling the Mysteries Behind Deep Pits Found Near Stonehenge Based on a comprehensive study, researchers are now convinced the shafts were human-made, likely dug during the Late Neolithic period roughly 4,000 years ago Sarah Kuta - Daily Correspondent December 10, 2025 9:59 a.m. The pits are evenly spaced around a large circle. University of Bradford In 2020, archaeologists in the United Kingdom made a surprising discovery. At Durrington Walls, a large Neolithic henge not far from Stonehenge, they found more than a dozen large, deep pits buried under layers of loose clay. The pits are mysterious. Each one measures roughly 30 feet wide by 15 feet deep, and together they form a mile-wide circle around Durrington Walls and neighboring Woodhenge. They also appear to be linked with the much older Larkhill causewayed enclosure, built more than 1,000 years before Durrington Walls. For the last few years, archaeologists have been puzzling over their origins: Were they dug intentionally by human hands? Were they naturally occurring structures, like sinkholes? Or is there some other possible explanation for the existence of these colossal shafts? Quick fact: The purpose of Durrington Walls While Stonehenge is thought to have been a sacred place for ceremonies, Durrington Walls was a place where people actually lived. In a new paper published in the journal Internet Archaeology, archaeologists report that they have a much better understanding of the pits’ purpose, chronology and environmental setting. And, now, they are confident the shafts were made by humans. “They can’t be occurring naturally,” says lead author Vincent Gaffney, an archaeologist at the University of Bradford, to the Guardian’s Steven Morris. “It just can’t happen. We think we’ve nailed it.” Chris Gaffney, an archaeologist at the at the University of Bradford, surveys the ground near Durrington Walls. University of Bradford For the study, researchers returned to the site in southern England and used several different methods to further analyze the unusual structures. They used a technique known as electrical resistance tomography to calculate the pits’ depths, and radar and magnetometry to suss out their shapes. They also took core samples of the sediment, then ran the soil through a variety of tests. For instance, they used optically stimulated luminescence to determine the last time each layer of soil had been exposed to the sun. They also looked for traces of animal or plant DNA. Astonishing' Stonehenge discovery offers new insights into Neolithic ancestors. Together, the results of these analyses indicate humans must have been involved, which suggests the pits could be “one of the largest prehistoric structures in Britain, if not the largest,” Gaffney tells the BBC’s Sophie Parker. Researchers suspect the circle pits were created by people living at the site over a short period of time during the Late Neolithic period roughly 4,000 years ago. They were not “simply dug and abandoned” but, rather, appear to have been part of a “structured, monumental landscape that speaks to the complexity and sophistication of Neolithic society,” Gaffney says in a statement. For example, the pits are fairly evenly spaced around the circle, which suggests their Neolithic creators were measuring the distances between them somehow. “The skill and effort that must have been required to not only dig the pits, but also to place them so precisely within the landscape is a marvel,” says study co-author Richard Bates, a geophysicist at the University of St Andrews, in a statement. “When you consider that the pits are spread over such a large distance, the fact they are located in a near perfect circular pattern is quite remarkable.” Researchers used multiple methods to investigate the pits at Durrington Walls. University of Bradford But who dug the pits? And, perhaps more importantly, why? Archaeologists are still trying to definitively answer those questions, but they suspect the shafts were created to serve as some sort of sacred boundary around Durrington Walls. Their creators may also have been trying to connect with the underworld, per the Guardian. “They’re inscribing something about their cosmology, their belief systems, into the earth itself in a very dramatic way,” Gaddney tells the BBC. Get the latest stories in your inbox every weekday.

Is red meat bad for you? Limited research robs us of a clear answer.

We’d all appreciate more definitive guidance. Eating a varied diet is a wise move while we wait.

Over and over, we ask the question: Is Food X good or bad for you? And, over and over, belief in the answer — whether it’s yes or no — is held with conviction totally out of proportion with the strength of the evidence.Today’s illustration: red meat. It has become one of the most-disputed issues in food. It’s so polarizing that some people decide to eat no meat at all, while others decide to eat only meat. It’s poison, or it’s the only true fuel.The latest salvo in the Meat Wars was kicked off by a new report that outlines the optimal diet for both people and planet. The EAT-Lancet Report comes down hard on red meat; its recommended daily intake is a mere 14 grams — that’s half an ounce.Read on, and the news gets worse: “Because intake of red meat is not essential and appears to be linearly related to higher total mortality and risks of other health outcomes in populations that have consumed it for many years, the optimal intake may be zero.”Note that word: “related.” It’s the source of the problem with the report and its recommendation.The EAT-Lancet report, by researchers from 17 countries, bases its recommendation solely on observational data. When you do that, meat comes out looking pretty bad. In study after study, people who report eating a lot of meat have worse health outcomes than people who eat little. Meat-eating correlates with increased risk of heart disease, some cancers and all-cause mortality.But, as always with observational research that attempts to connect the dots between diet and health, the key question is whether the meat itself, or something else associated with a meat-heavy lifestyle, is actually causing the bad outcomes.That’s a hard question to answer, but there are clues that people who eat a lot of meat are very different from people who eat a little.Let’s look at a study, published in JAMA Internal Medicine, cited by the EAT-Lancet report; it has a convenient demographic summary. According to it, people in the top one-fifth of meat eaters are different from people in the bottom fifth in a lot of important ways: They weigh more, they’re more likely to smoke, they’re not as well-educated, they get less exercise, and they report lower intakes of fruit, vegetables and fiber. On the plus side, they report drinking less alcohol. But other than that, we’re looking at a litany of markers for a lifestyle that’s not particularly health-conscious.So, to suss out whether it’s the meat that’s raising disease risk, you have to somehow correct for any of the differences on that list — and most of that information also comes from observational research, so even the confounders are confounded.Then there are the things you can’t correct for. Sleep quality, depression and screen time, for example, all correlate with some of the same diseases meat correlates with, but most studies have no information on those.All this confounding explains one of my all-time favorite findings from observational research. It comes from the same study the demographics came from (analyzed in a 2015 paper). Sure enough, the people who ate the most meat were more likely to die of cancer and heart disease, but they were also more likely to die in accidents. And the biggest difference came from the catchall category “all others,” which invariably includes causes of death that have nothing to do with meat.Basically, there’s a very simple problem with relying on observational research: People who eat a lot of meat are very different from people who eat less of it. The meat definitely isn’t causing the accidental deaths (unless, perhaps, they’re tragic backyard grill mishaps), and it isn’t causing at least some of the “all others” deaths, so we know that heavy and light meat-eaters are different in all kinds of ways.That’s where controlled trials come in.In a perfect world, we could figure this out by keeping a large group of people captive for a lifetime, feeding half of them meat, and seeing what happens. Okay, maybe that’s not a perfect world, but it would be the best solution to this particular problem.Instead, we have trials that are short-term (because of logistics and cost), and necessarily rely on markers for disease, rather than the disease itself. For that to be useful, you need a marker that’s a reliable indicator. For a lot of diseases — including cancer — those are hard to come by. For heart disease, we have a good one: low-density lipoprotein (LDL) cholesterol. So, most of the controlled trials of meat-eating focus on heart disease.If you spend some time reading those trials (and I did, so you don’t have to), you find that most of them show some increase in LDL cholesterol, although it’s generally small.A 2025 analysis of 44 controlled trials on meat found that the only ones showing positive cardiovascular outcomes had links to the meat industry, and even then, only about one in five came out positive. Of the independent studies, about three-quarters showed negative outcomes, and the remaining one-quarter was neutral.This isn’t surprising. Red meat contains saturated fat, and we have countless trials that demonstrate sat fat’s ability to raise LDL. But if the meat you eat is relatively lean, that effect is going to be small.The lesson here is that we don’t have a lot of good evidence on meat and health. The observational evidence is hopelessly confounded, and the evidence from clinical trials is woefully limited. There’s so much we simply don’t know. There may be other ways meat raises risk (leading to over-absorption of heme iron and stimulating the production of TMAO, or trimethylamine N-oxide), but there’s little definitive evidence for them. And, of course, there’s the question of what you eat instead. If you’re eating red meat instead of, say, instant ramen, that may be an improvement. If, instead, you’re cutting back on your lentils, not so much.As always, the single-most important thing to remember about nutrition is that what we know is absolutely dwarfed by what we don’t know. Which means that, if you’re making decisions based on what we do know, you could very well be wrong.So what’s an eater to do? Meat is a nutritious food. In fact, animal foods are the only natural sources of a vitamin we need — B12 — which is an indication that we evolved with meat and dairy as part of our diet. It’s very hard to know whether eating some lean meat leads to better outcomes than eating no meat, but I think some meat is a good hedge against all that uncertainty. (The ethical and environmental concerns are also important, but for today let’s focus on health.)But plant foods are also nutritious. And eating a wide variety of them is also a good hedge against uncertainty. Which means the carnivore diet — all meat, all the time! — is a pretty bad bet.Unfortunately, “uncertainty” is not a word that features prominently in the Meat Wars. Instead, we have an unappetizing combination of nastiness and sanctimony, with each camp convinced that the truth and the light are on their side.Not that this is a metaphor for our times or anything.

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